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Recent studies have shown that IL-4 and IL-13 weaken skin resistance to pathogens by reducing antimicrobial peptides, hence rendering skin more susceptible to infectious organisms (85)

The decreased immune response could be reversed by the administration of N-acetylcysteine (Atkuri et al., 2007

showed that TMAO not only promoted cellular inflammation but also accounted for oxidative stress, thus playing an indirect role in promoting expression of inflammatory cytokines (74)