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Acetylated glutathione then crosses the intestinal barrier, penetrates the cells, and then loses its acetyl group under the effect of cytoplasmic thioesterases

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The ER-resident GPx7 or GPx8 isoforms protect -cells of insulin-secreting INS against lipotoxicity by enhancing the antioxidant capacity of ER without compromising insulin production and oxidative protein folding mechanisms [19, 59]