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Semaglutide disrupts multiple steps in this chain

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e., infants born before full term) with low TH levels exhibit a higher likelihood of developing such defects, likely due to impaired cone development [6,7,8,9]

These cytokines disrupt the production of key ECM proteins, including collagen type II and aggrecan, while promoting the synthesis of matrix metalloproteinases (MMPs), which leads to ECM degradation and further progression of OA [2]

To find sources of heterogeneity and explore the effect sizes, we carried out subgroup analyses based on some confounding factors