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Concretely, APOC2-K70la stabilizes APOC2 and promotes extracellular lipolysis in NSCLC cell lines, thus leading to free fatty acid release, regulatory T-cell accumulation, immunotherapy resistance, and metastasis in NSCLC

Lysine biosynthesis and sulfur metabolism were not regulated by XJZ-A after XJZ subtracted the assistant drug, suggesting that the assistant drug plays a major role in the regulation of the above 2 metabolic pathways by XJZ

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Lastly, the significance of GpoA in virulence was demonstrated using a murine infection model

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