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Broad cytoprotective signaling, including ERK1/2 and FAKpaxillin activation, which enhance cell survival, adhesion, and motility at injury sites

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In experimental models, BPC-157 has been observed to accelerate wound healing , stimulate collagen synthesis , and enhance angiogenesis through upregulation of growth factors such as VEGF and modulation of the FAK-paxillin signaling pathway

BPC-157 is stable in gastric acid (unlike most peptides), which is one of its distinguishing pharmacologic properties 1