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Regulation of ubiquitination and degradation of p53 in unstressed cells through C-terminal phosphorylation

The differences we found between our conditions were produced by a one-time exposure to an online picture of a snus product with an HWL and may not be generalizable to real-life exposure or have any long-term impact

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Since IL-1 engages an IL-1 receptor on ILC3s and promotes their proliferation and IL-17A production 14 , these findings suggest that the smoking-induced sputum ILC3s and circulating memory-like CD4 + CD45RO + ILC3 subset in asthma patients could be generated by smoke-damaged airway epithelial cells rather than circulating immune cells