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It suppresses excessive reactive oxygen species (ROS) and maintains mitochondrial function by modulating the Receptor-Interacting Protein Kinase 3-Mixed Lineage Kinase Domain-Like protein (RIP3-MLKL) (necroptosis-related) and Receptor-Interacting Protein Kinase 1 (RIP1)-Nuclear-related factor 2 (Nrf2)-Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-B) (antioxidant/inflammatory) pathways, ultimately promoting cellular survival

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[139] Pigatto PDM, Brambilla L, Ferrucci S, Guzzi G

In IBD, dysbiosis has been linked to an increased production of pro-inflammatory cytokines, which may influence both intestinal and brain inflammation (Shaikh et al., 2023)