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During glucose deprivation, astrocytes repurpose lipolytic fragments into lipid droplets via diacylglycerol acyltransferase (DGAT)-catalyzed esterification and perilipin-2 (PLIN2)-mediated stabilization, sequestering cytotoxic fatty acids to protect neurons from lipotoxicity [21,22,23,24,25,26,27,28,29,30,31,32,33,34,35,36] (Table 1)