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A., Johannsson, G

Transl Psychiatry 13 , 58 (2023)

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Fe II binding to Nha3 followed by GTP binding then stimulates GTP hydrolysis and Fe II transfer to the -subunit

However, the dual activity of KYNA, antagonizing NMDARs and 7nAChRs both of which are critical for normal brain function complicates pharmacological targeting, as achieving therapeutic modulation within an optimal window risks unintended and potentially severe side effects