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W.HiguchiM.KumagaiS.OhnoH.et al (2013)

2019;47(W1):W40207

Strom, P

Unlike endocrine peptides, BPC-157 does not operate through hormone receptor pathways, instead exerting its effects through localised cellular signalling and molecular repair mechanisms

290 With aging, mitochondrial dysfunction and genomic instability can lead to the buildup of oxidative stress and DNA damage, which can disrupt energy production and genomic maintenance in motor neurons, processes implicated in ALS pathogenesis