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Disruption of mitochondrial complex I and metabolic stress can also promote necroptosis, mediated by RIPK3-dependent pathways observed in Salmonella and Listeria infections (Sai et al., 2019

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Our study showed that HepG2 cells, which primarily rely on extracellular cystine as a source of cysteine 20 , responded to cystine deprivation by reducing anabolic signaling including attenuation of mTORC1 pathway and activation of the ISR (Fig

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Inhibition of atherosclerosis progression by modular micelles