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Fluoxetine further enhances NIX (BNIP3L)-mediated mitophagy in the hippocampus of CUMS mice [127, 128] and promotes autophagic flux and mitophagy in astrocytes via a p53-dependent mechanism, facilitating the clearance of damaged mitochondria [101]

It plays a pivotal role in contributing to global morbidity and mortality rates

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GSH abolished this inhibitory effect and promoted the recovery of SLC7A11 and GPX4 levels (Fig