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Moreover, genetic studies should be focused on an individual selenium intake in order to identify genegene and geneenvironment interactions responsible for stroke development in hypertensive subjects

While the protection of normal cells against radiation-induced cell death by ebselen has been attributed to its ability to detoxify cellular/mitochondrial ROS through the GPx mechanism, the molecular mechanism contributing to its radio-sensitization effect in cancer cells is still not fully understood

The design strategy and mechanism were outlined in Fig

Furthermore, ATF4, under conditions of stress, has been observed to interact with mTORC, thereby influencing cell survival