doi: 10.3389/fneur.2017.00333 Received 26 April 2017 Accepted 26 June 2017 Published 20 July 2017 Volume 8 - 2017 Edited by Vittorio Maglione, IRCCS Neuromed, Italy Reviewed by Jenny Sassone, Vita-Salute San Raffaele University, Italy
Those include increasing glutathione levels and increasing immune support
De-escalation of treatment in HER2positive breast cancer: determinants of response and mechanisms of resistance
In a mouse model of stroke, intracerebroventricular sodium selenate treatment elevates GPX4 levels by activating transcription factors activating enhancer binding protein 2 and specificity protein 1, while also providing protection against excitotoxicity and endoplasmic reticulum stress-induced cell death independent of GPX4 (102)
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