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A published report has shown that the NF-B member, RelB, plays a role in upregulating GPX4 expression and suppressing ferroptosis, thereby increasing the resistance of ER+ breast cancer cells to Tamoxifen [32]

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Mitochondria-targeted antioxidants, including MitoQ and SS31, may serve as potential therapeutic agents that reduce free radicals, maintain mitochondrial quality and function, enhance autophagy and mitophagy, and help cells remove damaged mitochondria [24, 31]

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