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Abstract Non-alcoholic fatty liver disease (NAFLD) is characterized by core pathological features such as hepatic lipid accumulation, oxidative stress, and inflammatory responses

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Moreover, the nuclear receptor coactivator 4 (NCOA4)-mediated selective autophagy pathway (ferritinophagy) increases cellular labile iron pool levels to promote the rapid intracellular accumulation of ROS, which is critical for ferroptosis [61]

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While topical treatments can provide some visible improvement, addressing pigmentation from within often provides more sustainable and long-term results