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glutamate) where thought to be the primary mechanism for these drugs but now neuroplasticity can also require presynaptic axonal 5-HT2A activation

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However, future research is needed to unravel the biological mechanisms underlying this clinical observation

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Additionally, it has been suggested to inhibit the aggregation of proteins associated with neurodegenerative diseases, potentially slowing disease progression

[Scientific Research] Perna AF, Ingrosso D