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Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired

Medically reviewed by Veronica Hackethal | MD, MSc , Harvard University | University of Oxford | Columbia Vagelos College of Physicians and Surgeons on April 23rd, 2026

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2018;65:e12508

This review summarizes the mechanisms of CagA, VacA, NapA, SodB, gGT, LPS and OipA or H