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While our experiments illustrate the ability of IH to negatively regulate Tbr2 + INPs, our data suggest that IH exposure with durations greater than ten days is required to see evidence of pro-neurogenesis signaling within early neuronal progenitor populations

Related mode-of-action work has evaluated changes in body weight, fat mass, lean mass, and feeding behavior in controlled models, helping researchers explore how amylin pathway activation reshapes whole-body metabolic phenotypes [4]

it requires targeted agents to actively signal and promote repair

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