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Microarray and RNA-seq investigations of cells exposed to GHK-Cu identified broad patterns of gene expression changes affecting hundreds or thousands of genes in some studies touching on collagen and extracellular matrix genes, antioxidant defense genes, DNA repair genes, immune-related genes, and others

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2 These include: A ME/CFS-like phenotype with post-exertional malaise and impaired bioenergetics A dysautonomia/POTS-dominant presentation A mast celldriven inflammatory phenotype Persistent cardiopulmonary injury This heterogeneity explains why a single treatment approach fails

This cascade results in irreversible proteotoxic stress, ultimately leading to cardiomyocyte death [74]

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