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Among antioxidants whose presence has been documented in the thyroid, peroxiredoxins (Prxs) are of a special significance, as they are involved in the process of H 2 O 2 elimination, when this ROS is formed in response to TSH, and they protect thyroid cells from H 2 O 2 -induced apoptosis [130, 131]

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The double increase in Asc in ischemic tissue compared to healthy tissue indicates increased ROS formation under ischemic conditions and confirms the role of oxidative stress in ischemia

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