ghk-cu endogenous levels decline with age Frontiers Are We Ready to Measure

FOXO4-DRI's selectivity claims are from cell and animal models

Published trial abstracts can also be misunderstood

Hostynek, J

Its believed to promote musculoskeletal repair by upregulating growth factors, stimulating the formation of new blood vessels, or modulating nitric oxide synthesis, but no mechanism of action has been proven.34 TB4, however, has known mechanisms by which it speeds recovery from injury: inhibiting inflammation, preventing cell death, and (as suspected with BPC) promoting blood vessel formation.35 On top of the benefits for physical recovery, the ability to form new blood vessels points to promising implications for cardiovascular health

PMID: 32050436

This combination could result in more efficient repair, which in turn may deepen sleep further