The combination of dysregulated lipid homeostasis and impaired purine metabolism may offer a mechanistic explanation for the similar symptoms observed in ME/CFS and GWS
,, Although there are several inhibitors of the cystine/glutamate transporter other than sulfasalazine, these drugs have limitations of low bioavailability and low selectivity to of the cystine/glutamate transporter in vivo
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Both require refrigeration after reconstitution, but GHK-Cu also demands light protection due to coppers photosensitivity
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