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Further studies are warranted to investigate the basis for the relationship between these redox parameters and other biomarkers of oxidative stress, and to investigate the mechanisms underlying the association of anemia and excessive adiposity with alterations in the redox status in adolescence

DATS inhibited bacterial lipopolysaccharide (LPS)-induced macrophage activation by limiting LPS binding to toll-like receptor 4 (TLR4) and blocking the upregulation of TLR4 and TLR4-associated molecule MyoD88 expression (37)