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Reevaluation of the roles of luteinizing hormone and follicle-stimulating hormone in the ovulatory process

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Individuals with genetic variants associated with reduced GST activity or impaired antioxidant enzyme function may require higher glutathione support or more frequent dosing schedules

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Authors proposed a model wherein PL enters the cell, is hydrolyzed in its active form (hPL) with subsequent formation of a hPL:GSH complex that binds the active site of GSTP1-1 causing its inhibition (Fig