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This cascade restores PINK1/Parkin-dependent mitophagy and concurrently activates the NRF2 pathway, enhancing antioxidant responses, like NQO-1and HO-1, and consequently reducing oxidative stress in ectopic lesions

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These may result in oxidative stress in which molecular signaling is altered

6 Conclusions and prospects Hitherto, it is clear that innate immunity holds a pivotal position in the development of autoimmune diseases

in human arteries