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Nonetheless, there remains an ongoing need for additional medicines, particularly those with a distinct mode of action that could be used independently or in combination with the current incretin-based therapeutics

documentation of both conditions in the same encounter is sufficient

Both are pro-angiogenic: BPC-157 through VEGFR2 sensitization and the AkteNOS axis, [7] thymosin beta-4 through its actin-binding LKKTET motif and downstream VEGF-linked signaling

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