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It has been proposed that stressful or traumatic incidents in genetically susceptible individuals may trigger the development of ME/CFS symptoms (58, 61)

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Wild type SOD1 can be misfolded by the aberrant translation, post-translational modification or oxidative modifications, due to the pathological conditions of the CNS environment, and produce toxic insoluble aggregates, as mutated SOD1 (Bosco et al., 2010

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