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We have shown that the stimulation of GSH efflux by verapamil, a MRP1 ligand 11 , triggers the selective cell death of BHK21 (Baby Hamster Kidney 21) cells overexpressing MRP1 following a rapid and massive depletion of cellular GSH 12

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Viral spike proteins, persistent inflammatory cytokines, and circulating autoantibodies create a highly toxic environment that impairs the enzyme responsible for making NO, known as endothelial nitric oxide synthase (eNOS)