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In vitro assays delineated MSU crystals propensity to induce TH1 cells to overexpress miRNA-146a, implying that miRNA-146a acted as a transcriptional brake in monocytes, which was suppressed during acute inflammatory responses to MSU crystals

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Researchers proposed that BPC-157s anti-inflammatory properties and its ability to stimulate angiogenesis may create conditions more favorable to neuronal repair in the post-injury environment

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