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Further, this study provides new evidence that mitochondrial superoxide production may be elevated in certain brain regions and that a neuroinflammatory process may promote oxidative stress and damage in affected cells

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Following VEGFR2 upregulation and internalization, BPC-157 time-dependently activates the downstream VEGFR2-Akt-eNOS signaling cascade

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These reductions translated into enhanced locomotor activity and lifespan extension in treated flies [44]