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These findings, in line with several recent studies reporting the protective effects of statins in various kidney diseases [42,43,44,45], underscore the critical role of lipid metabolic dysregulation in CaOx-induced renal injury and suggest that pharmacological modulation of lipid metabolism may serve as a complementary strategy to targeting the PRMT1/UBE2m axis
Similar to MB, we also observed that TB and NR increased cellular oxygen consumption and decreased lactate production although the action and pattern of NR on ECAR was different from that of MB and TB
doi: 10.1016/j.prp.2023.155024 125 GesIAPereiraMRMCrottiEPereiraGPYoshitaniMMCastroMAet al
Accumulating clinical and experimental evidence confirms ferroptosis as a pivotal driver of kidney disease onset and progression
The total flinching numbers of the BPC-157 and morphine groups were significantly decreased in phase 1, but those of the BPC-157 groups, except the morphine group, were not different from the control group (Fig
