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The resulting death of pericytes leaves the microvasculature continuously constricted, leading to microcirculatory failure and long-term cerebral hypoperfusion due to the contractile apparatus remaining in rigor upon pericyte death, thus reducing capillary diameter [94, 104]

Using transcriptomic data of non-neurological brains, we identified genes (e.g., SNCA , SCARB2 , and ZNF184 ) and modules of co-expressed genes for which the expression decreased or increased across brain regions defined by the Braak ascending scheme

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If you're suddenly breaking out on your cheeks when you never break out there, that's not purging

The effect is on cell programming, not on a continuously triggered receptor cascade