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10.3389/fonc.2021.760971 [DOI] [PMC free article] [PubMed] [Google Scholar] Xu Y., Zhao W., Xu J., Li J., Hong Z., Yin Z., et al
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In advanced CKD, the dysregulation of mineral metabolism and PTH overproduction drive indolent osteoblasts into hyperfunction, which disrupts the normal bone remodeling process and results in the loss of both bone quality and quantity [101]
Because oral glutathione supplements have historically demonstrated poor bioavailability and are rapidly broken down by enzymes in the gastrointestinal tract, the body relies heavily on intracellular de novo synthesis to maintain its protective antioxidant pools
Evidence from GH-transgenic mice
Therefore, genetic ablation or pharmacological inhibition of Parp-1 enhances the Sirt1 activity through restoring the NAD + content, providing a protection benefit for various tissues, including the liver, muscle and brown adipose tissue
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