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Linking lipid metabolism to the innate immune response in macrophages through sterol regulatory element binding protein-1a

Mitochondrial Ca 2+ overload is closely associated with mPTP opening, dissipation of m, and impairment of oxidative phosphorylation, thereby placing bioenergetic failure among the earliest direct consequences of abnormal ERmitochondria Ca 2+ signaling

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Oral glutathione is largely broken down by stomach acid, significantly reducing bioavailability and effectiveness

10.1017/S0031182007002508 Parasitology 34 MarcoglieseD

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