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These findings collectively emphasize that gut dysbiosis, increased intestinal permeability, and even gutlung microbial translocation can amplify neuroinflammation and worsen ischemic stroke outcomes, underscoring the need to consider the gutbrainlung axis as a potential target for improving stroke prognosis

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Direct neuronal protection by the proteaseactivated receptor PAR4 antagonist ML354 after experimental stroke in mice

Some of these methods have already been shown to increase the delivery of intact peptides and other substances such as chemotherapy agents, biological markers, cellular ligands and receptors, and other substances to their intended targets in the body, thereby increasing overall bioavailability and effectiveness