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doi: 10.1105/tpc.104.029603 53 JohnstonE

Together, these findings reveal that global GPR146 deficiency leads to suppression of hepatic PPAR signaling and its downstream adipogenic targets, along with reduced expression of inflammatory and ECM remodeling genes

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Current research indicates that neurotrophic factor pathway modulation serves as the primary driver of its therapeutic effects, supported by at least four additional mechanisms that work synergistically to promote neuronal survival, plasticity, and functional recovery