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Overall, our GLP-1 thesis is still sound and right for the right reasons

Research on brainstem mechanisms of amylin-induced anorexia confirmed that amylin-responsive and GLP-1-responsive neurons are largely separate populations

It is one of the most researched synthetic peptides in preclinical studies, examined for its interaction with nitric oxide pathways, growth factor signaling, and other cellular communication systems

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This inhibits K + influx, and therefore favours K + efflux through the K ATP -sensitive channel during GSIS [96]