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To address this, the lens employs a unique microcirculation system sustained by Na + /K + -ATPase pumps, aquaporins, and gap junctions that generate circulating ionic currents [6]

Elevated serum cholesterol, liver damage, systemic inflammation, and hypoxic conditions can increase XO secretion via hepatic and other visceral pathways into the bloodstream and bind to the endothelial cell surface, initiating the overproduction of superoxide [77]

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40,41 These oxidative modifications of cysteine can be reverted to the free thiol state (-SH) by specific reductants