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Aging Cell , 22(4), e13759

This deactivation of MKPs allows for the unimpeded progression and sustained activation of the pro-inflammatory ERK1/2, JNK, and p38 signaling cascade within the MAPK pathway (Son et al., 2011)

Cong et al

Although further clinical evidences are still needed for elucidating the precise mechanism, it is considered plausible that EAAC1 dysfunction leading to GSH depletion is closely involved in neurodegenerative diseases

101 KDM5A inhibition increases MPC1 expression and reduces the susceptibility of erlotinib-tolerant persistent head and neck cancer cells (erPCCs) to ferroptosis inducers

Thus, research findings obtained so far suggest the differential action of ebselen in cells of normal and cancerous organs