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Once PL-PUFAs are incorporated into the membrane environment, iron-dependent enzymes and free iron utilise molecular oxygen (O 2 ) to catalyse peroxidation reactions, generating PL-PUFA-OOH

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Conversely, some mechanisms that inhibit LIP expansion have been discovered

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Thus, we have demonstrated that GSH-targeted therapy selectively kills PC cells in an oxidative stress-dependent manner and that the differences in basal ROS level and antioxidant scavenging capacity induced by stratified expression of VRK2 can well account for the heterogeneous sensitivity to GSH inhibition

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