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Alpha phellandrene significantly decreased the levels of pro-inflammatory cytokines such as TNF- in the alpha phellandrene treated group

First, while the rescue experiments with ferric citrate and NAC strongly support an iron deficiency and ROS-mediated mechanism, the absence of established positive controls, such as the iron chelator deferoxamine (DFO) or direct ROS generators, precludes a definitive comparative conclusion that the observed effects are uniquely attributable to CGAs iron-chelating activity

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& Adachi, H

Cell adhesion monitoring on xCELLigence has been previously demonstrated and can be assessed in short time experiments [13, 14]