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Taken together, the progressive elucidation and investigation of ALS-related disease-causing genes, the successful construction of disease models through the utilization of technologies such as exon sequencing and gene editing, and the further investigation of the potential molecular regulatory mechanisms between ALS mutant genes and mitochondria will facilitate the opening of a new avenue for the study of targeted therapy for ALS disease
Qi W, Li Z, Xia L, Dai J, Zhang Q, Wu C, Xu S (2019) LncRNA GABPB1-AS1 and GABPB1 regulate oxidative stress during erastin-induced ferroptosis in HepG2 hepatocellular carcinoma cells
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Arabidopsis thaliana glutamate-cysteine ligase: functional properties, kinetic mechanism, and regulation of activity
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