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Crucially, the ROS generated during GSH degradation by CHAC1 may also cause further TP53 mutations, creating a vicious self-reinforcing cycle: CHAC1 activity promotes the very TP53 mutations that allow the tumor to exploit CHAC1s harmful effect for growth

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MVA has the capacity to deliver multiple heterologous antigens and the ability to trigger both cellular and humoral immunity 35,36