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In addition, classical ferroptosis activators (such as ML-210) or inhibitors (such as ferrostatin-1, Fer-1) modulate intracellular autophagic flux in testicular injury

In theory, this could include promoting the growth of existing tumours or disrupting normal endocrine function

When these pro-inflammatory mediators reach the hypothalamus, they increased the levels of oxidative stress and neuronal inflammation in the hypothalamus through processes such as microglia proliferation (124, 125)

It plays a critical role in detoxification, immune support, and cellular repair

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