Finally, we outline future research directions, offering new insights for both basic experimental and clinical studies aimed at treating uterine fibroids
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(2001) Morphological changes in dendritic spines associated with long-term synaptic plasticity
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Metabolic changes of lipids in non-alcoholic fatty liver disease Lipolysis of adipose tissue Most NAFLD patients have increased white adipose tissue (WAT) mass and its expansion is a high-risk factor in the progression of NAFLD [12, 13]
In septic models, ATF4 depletion in Ly6C+ monocyte precursors (P1 cells) disrupts gMac (P4 cell) differentiation coupled with aberrant proliferation and increased apoptosis of P1 cells, leading to exacerbated intestinal barrier dysfunction (132)