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The combination of NADPH and Mito-apocynin, a mitochondria-specific NADPH oxidase (NOX) inhibitor, effectively reversed KA-induced neuronal damage and the down-regulation of TP53-induced glycolysis and apoptosis regulator and NOX4 [122]

retain patient-specific genetic and developmental constraints allow neurons to self-organize into functional networks reveal disease-associated dynamics without behavioral confounds What is MEA

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The amylin receptor agonism it produces slows gastric motility specifically during the postprandial window

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Then read the purity and identity lines