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Lipid hydroperoxides accumulate in the cellular membranes of lung cells, destabilizing the barrier functions of alveolar epithelial and endothelial cells

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doi: 10.3390/pharmaceutics16030397

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Ultimately LC, CFS, and low flow POTS seem to stem from antioxidant insufficiency (redox imbalance) and mitochondrial dysfunction

Additionally, the exploration of whether the drug affects Th17 cell activity by influencing STAT3 activity remains insufficiently thorough