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However, we advocate for further research, particularly focusing on these parameters with further studies on liver enzymes and serum electrolytes, to deepen our understanding of the complexities surrounding EMF exposure and its potential health implications

CHAC2-mediated glutathione metabolic reprogramming drives N1 polarization of bone marrow neutrophils and exacerbates inflammatory comorbidities

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Other pharmaceuticals within this category, such as etoposide and camptothecin, exhibit similar senescence-inducing properties to doxorubicin [6]

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Tissue damage was graded on a scale from 0 to 10, where 0 indicated no observable injury and 10 represented the highest degree of organ damage