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Other suggested mechanisms include inhibiting the activation of the downstream adapter protein and also the epidermal growth factor receptor in human skin cancer cells (A431) [302]

In addition, levels of Toll Like Receptor 4 (TLR4) and inflammatory factors, such as Nuclear Factor B (NF-B), IL-6 and Monocyte Chemoattractant Protein-1 (MCP-1), were also significantly decreased compared to the untreated PE group

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glutathione S-aralkyltransferase